Preliminary reports point out conolidine may well inhibit unique ion channels, minimizing neuronal excitability and limiting discomfort signals. This system is especially pertinent in neuropathic suffering, where irregular signaling causes persistent soreness. Moreover, conolidine seems to influence G protein-coupled receptor (GPCR) pathways integral to ache perception. Computational ... https://toddg284zqi9.signalwiki.com/user